A retrograde GCN2/eIF2α, but ATF4 independent, mechanism maintains synaptic strength under acute amino acid scarcity at the NMJ https://www.biorxiv.org/content/10.1101/2022.11.03.515133
A retrograde GCN2/eIF2α, but ATF4 independent, mechanism maintains synaptic strength under acute amino acid scarcity at the NMJ https://www.biorxiv.org/content/10.1101/2022.11.03.515133